Multiple roles for interbacterial T6S
Alistair B. RussellAlistair B. in 2009 from Cornell University, New York,
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places that sell cards against humanity, Seattle, USA. His research in the Mougous laboratory has focused on the distribution and activities of bacteria targeting T6SS effectors.S. Brook PetersonS. in 2008 in the laboratory of Jo Handelsman at the University of Wisconsin Madison, Wisconsin,
cards against humanity price?, USA. Following graduation, she moved to Seattle, Washington,
cards against humanity game, USA, to pursue postdoctoral work in the laboratory of Matthew Parsek. In 2013 she joined the Mougous laboratory as a senior scientist. Her research interests concern the networks of interactions among bacterial species in the environment.Joseph D. MougousJoseph D. in 2004 in the laboratory of Carolyn Bertozzi at the University of California, Berkeley, USA. He subsequently undertook his postdoctoral training in the laboratory of John Mekalanos at Harvard Medical School, Boston, Massachusetts, USA. In 2007, he started his own laboratory at the University of Washington, Seattle, USA, where he is currently an associate professor in the Department of Microbiology. With a focus on the type VI secretion system, his research is concerned with the mechanisms by which bacteria influence each other and the impact of bacterial interactions on microbial communities and human health. Joseph D. Mougous homepage.Contact Joseph D. MougousType IV secretion systemsSecretion systems involved in the transfer of both DNA and proteins to bacterial and eukaryotic targets.(VgrG). A substrate and structural component of the type VI secretion system (T6SS). and specific immunity determinants that protect the producing cell are typically encoded by adjacent genes.(Rhs proteins). Large multi domain proteins that have a central element consisting of a repeating motif and a toxin domain at their carboxyl terminus. Systems that consist of a toxic element and an unstable cognate antitoxin that mediate toxicity in the producing cell such that depletion of the antitoxin results in death or senescence of the organism.(VBNC). is not necessarily mutually beneficial.